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Consequences physiologiques ironman
Publié : 15 mars 2009, 19:24
par llp
Bonjour à tous,
je recherche des articles ou eventuellement des passages d'ouvrages pouvant me renseigner sur les suites physiologiques (blessures, pertes d'oligo-éléments, traumatismes
à court mais aussi à long terme...) de triathlons longue distance..
Merci à tous.
Mon adresse mél:
lapompe63@hotmail.com
Re: Consequences physiologiques ironman
Publié : 15 mars 2009, 20:21
par lullabies
comme tous les sports, le tri c'est bon pour la santé
comme tous les sports, quand t'en fait trop t'es grillé
biz
Re: Consequences physiologiques ironman
Publié : 15 mars 2009, 20:57
par ironturtle
Clair, net, sobre, précis.

Re: Consequences physiologiques ironman
Publié : 15 mars 2009, 21:33
par Ben boost
ironturtle a écrit :Clair, net, sobre, précis.

Ouaips, il change le gamin.

Re: Consequences physiologiques ironman
Publié : 15 mars 2009, 21:44
par -Antoine-
Personnellement, je trouve que le marathon d'un IM est au final moins cassant au niveau articulaire qu'un marathon sec
Re: Consequences physiologiques ironman
Publié : 15 mars 2009, 22:00
par Meish
Forcément, sur les 42km les mecs en font à peine 30 en courant

Re: Consequences physiologiques ironman
Publié : 16 mars 2009, 00:17
par aurelie.218
-Antoine- a écrit :Personnellement, je trouve que le marathon d'un IM est au final moins cassant au niveau articulaire qu'un marathon sec
Pourquoi pourquoi?
Ca permettrait de me rassurer

Re: Consequences physiologiques ironman
Publié : 16 mars 2009, 01:44
par pierre-yves
Adapations à l'entraînement... à mon avis faire un ironman en étant bien préparé est moins traumatisant qu'un surentraînement dans n'importe quel sport.
Ici l'auteur montre que le triathlète s'est adapté à la production de radicaux libres (stress oxidant) et que ses défenses naturelles lui permettent de surmonter celle de l'IM.
Neubauer, O., et al., No indications of persistent oxidative stress in response to an ironman triathlon. Med Sci Sports Exerc, 2008. 40(12): p. 2119-28.
INTRODUCTION: Training for and competing in ultraendurance exercise events is associated with an improvement in endogenous antioxidant defenses as well as increased oxidative stress. However, consequences on health are currently unclear. PURPOSE: We aimed to examine the impact of training- and acute exercise-induced changes in the antioxidant capacity on the oxidant/antioxidant balance after an ironman triathlon and whether there are indications for sustained oxidative damage. METHODS: Blood samples were taken from 42 well-trained male triathletes 2 d before an ironman triathlon, then immediately postrace, 1, 5, and 19 d later. Blood was analyzed for conjugated dienes (CD), malondialdehyde (MDA), oxidized low-density lipoprotein (oxLDL), oxLDL:LDL ratio, advanced oxidation protein products (AOPP), AOPP:total protein (TP) ratio, Trolox equivalent antioxidant capacity (TEAC), uric acid (UA) in plasma, and activities of superoxide dismutase (SOD), glutathione peroxidase (GSH-Px), and catalase (CAT) in erythrocytes. RESULTS: Immediately postrace, there were significant increases in CD, AOPP, TEAC, UA (for all P < 0.001), and AOPP:TP (P < 0.01). MDA rose significantly (P < 0.01) 1 d postrace, whereas CD (P < 0.01), AOPP (P = 0.01), AOPP:TP (P < 0.05), and TEAC (P < 0.001) remained elevated. OxLDL:LDL trended to increase, whereas oxLDL significantly (P < 0.01) decreased 1 d postrace. Except for GSH-Px (P = 0.08), activities of SOD (P < 0.001) and CAT (P < 0.05) significantly decreased postrace. All oxidative stress markers had returned to prerace values 5 d postrace. Furthermore, several relationships between training status and oxidative stress markers, TEAC, and antioxidant enzyme activities were noted. CONCLUSIONS: This study indicates that despite a temporary increase in most (but not all) oxidative stress markers, there is no persistent oxidative stress in response to an ironman triathlon, probably due to training- and exercise-induced protective alterations in the antioxidant defense system.
Neubauer, O., D. Konig, and K.H. Wagner, Recovery after an Ironman triathlon: sustained inflammatory responses and muscular stress. Eur J Appl Physiol, 2008. 104(3): p. 417-26.
Ultra-endurance exercise, such as an Ironman triathlon, induces muscle damage and a systemic inflammatory response. As the resolution of recovery in these parameters is poorly documented, we investigated indices of muscle damage and systemic inflammation in response to an Ironman triathlon and monitored these parameters 19 days into recovery. Blood was sampled from 42 well-trained male triathletes 2 days before, immediately after, and 1, 5 and 19 days after an Ironman triathlon. Blood samples were analyzed for hematological profile, and plasma values of myeloperoxidase (MPO), polymorphonuclear (PMN) elastase, cortisol, testosterone, creatine kinase (CK) activity, myoglobin, interleukin (IL)-6, IL-10 and high-sensitive C-reactive protein (hs-CRP). Immediately post-race there were significant (P < 0.001) increases in total leukocyte counts, MPO, PMN elastase, cortisol, CK activity, myoglobin, IL-6, IL-10 and hs-CRP, while testosterone significantly (P < 0.001) decreased compared to prerace. With the exception of cortisol, which decreased below prerace values (P < 0.001), these alterations persisted 1 day post-race (P < 0.001; P < 0.01 for IL-10). Five days post-race CK activity, myoglobin, IL-6 and hs-CRP had decreased, but were still significantly (P < 0.001) elevated. Nineteen days post-race most parameters had returned to prerace values, except for MPO and PMN elastase, which had both significantly (P < 0.001) decreased below prerace concentrations, and myoglobin and hs-CRP, which were slightly, but significantly higher than prerace. Furthermore, significant relationships between leukocyte dynamics, cortisol, markers of muscle damage, cytokines and hs-CRP after the Ironman triathlon were noted. This study indicates that the pronounced initial systemic inflammatory response induced by an Ironman triathlon declines rapidly. However, a low-grade systemic inflammation persisted until at least 5 days post-race, possibly reflecting incomplete muscle recovery.
Re: Consequences physiologiques ironman
Publié : 16 mars 2009, 01:49
par pierre-yves
et ici les effets d'un surentraînement (5x2h de càp pendant 60 semaines chez des personnes ne s'étant jamais entraînée avant) provoquant une baisse des hormones sexuelles... on s'entend que l'étude est un peu dingue (en Iran il ne connaissent pas les comités d'éthiques) mais les résultats son intéressant.
1. Safarinejad, M.R., A.A. Kolahi, and K. Azma, The Effects of Intensive, Long-Term Treadmill Running On Reproductive Hormones, Hypothalamus-Pituitary-Testis Axis, and Semen Quality: A Randomized Controlled Study. J Endocrinol, 2008.
Effects of intensive exercise on hypothalamus-pituitary-testis (HPT) axis remain controversial. Our aim was to determine the effects of intensive, long-term treadmill running on reproductive hormones, HPT axis, and semen quality. A total of 286 subjects were randomly assigned to moderate-intensity exercise [~ 60% maximal oxygen uptake (VO2 max); group 1, n=143], and high-intensity exercise (~ 80% VO2 max, group 2; n=143) groups. The two groups exercised for 60 weeks in five sessions per week, each session lasting 120 min. This was followed by a 36-week low-intensity exercise recovery period. All subjects underwent routine semen analysis. Blood samples were drawn for the determination of the levels of the following hormones: luteinizing-hormone (LH), follicle-stimulating hormone (FSH), prolactin (PRL), testosterone (T), free testosterone (fT) inhibin B, and sex hormone binding globulin (SHBG). The HPT axis was assessed using gonadotropin releasing hormone (GnRH) and human chorionic gonadotropin (hCG) tests. After 24 weeks of exercise, subjects exercising with high-intensity demonstrated significantly declined semen parameters compared those exercising with moderate-intensity (P=0.03). Serum T, and fT, were begun to decrease, and serum SHBG, was begun to increase at the end of 12-week with both moderate and high-intensity exercises. The serum LH, and FSH concentrations decreased below the baseline level at 12-week in both groups (P=0.07 in group 1, and P=0.03 in group 2). Both groups had blunted LH and FSH responses to GnRH. These parameters improved to their pre-exercise level during the recovery period. Long-term strenuous treadmill exercises (overtraining syndrome) have a deleterious effect on reproduction.
Re: Consequences physiologiques ironman
Publié : 16 mars 2009, 10:54
par -Antoine-
aurelie.218 a écrit :-Antoine- a écrit :Personnellement, je trouve que le marathon d'un IM est au final moins cassant au niveau articulaire qu'un marathon sec
Pourquoi pourquoi?
Ca permettrait de me rassurer

Parce que tu mets en moyenne (quand tu es bien préparé) 27' de plus sur marathon IM/marathon sec... et que tu cours donc moins vite donc moins de chocs pour les articulations, foulée plus rasante, fréquence cardiaque plus basse,...
Re: Consequences physiologiques ironman
Publié : 19 mars 2009, 23:21
par triathlonnature
llp a écrit :Bonjour à tous,
je recherche des articles ou eventuellement des passages d'ouvrages pouvant me renseigner sur les suites physiologiques (blessures, pertes d'oligo-éléments, traumatismes
à court mais aussi à long terme...) de triathlons longue distance..
Merci à tous.
Mon adresse mél:
lapompe63@hotmail.com
Je n'en connais pas.
Mais le meilleur reste quand même son médecin traitant vu que l'on a l'habitude, même les pudiques, de se mettre nu devant lui.
Sinon, il est possible que tu ne puisse pas retrouver "ton" sommeil pendant quelques temps.
Une certaine démoralisation, normale, pour cela programmer une compétition dans le mois n'est jamais inutile.
Tout autre conseil est fonction de ton gabarit, ton entraînement, la faculté de récupérer et aussi le fait de sortir le mauvais de ta tête, beaucoup de chose en clair.
Une dernière chose : il ne faut pas avoir honte d'aller voir son médecin, tu n'es pas le premier.